Here's how you can make a difference

 

 

 

We have provided you with information that indicates that (1) consuming cooked foods strongly decreases your life expectancy, and that (2) a high calcium diet increases osteoporosis risk, but how can we bring this information to the general public?

By proving that we are right. When tests have shown that we are right, there are always media that will bring the truth into the open. If we manage to do so, we can save millions of lives. 

 

Luckily, it is very easy to do the required tests. 

Students can do these tests, to low execution costs.

 

What do you need? ((1) and (2))

 

- room for a few cages.

- young mice, post weaning

- mice feedings

- 4 years of feeding the mice daily

- analysis of the mice after they have died naturally. Of course, the results need to be adjusted for hormone levels, locomotor activity and body weight.

 

 

 

(1) The test "cooked versus raw foods regarding life expectancy"

This test is very simple; all you need are two groups of mice. One group is fed raw meat, raw egg yolks, raw nuts and fruits only (and drinking low mineral bottled water), while the other group is fed the same foods, but always cooked.

You can, of course, create a third group; to create 2 'cooked foods'-groups: one 'medium' and one 'well done'. (the feedings are cooked shortly and thoroughly, respectively)

All you need to do, is to register how long each mouse lives.

Of course, you can also have autopsy performed on each mouse.  

 

 

 

(2) The test "the influence of lifetime calcium intake on bone health"

Please check the Q and A regarding this test.

In this test you need at least 3 groups of mice. 

The diet of each group contains a different amount of calcium, varying from very little to very much calcium. 

The feedings in this test take a bit more work, because all groups need to consume the same feedings, with calcium contents as the only variable. 

All mice are fed dehydrated beef and fruits only, and drinking water. (no nuts because of their high calcium contents)

Calcium will be added to the drinking water of all mice except for the 'low-calcium' group, differing per group.  The drinking water needs to be low-mineral water, like Volvic, El Dorado or Montcalm.

After the mice have died naturally, their bones need to be examined for porosity (porosity is NOT about bone density, but about voids that undermine the structure of the bone, making it weaker regardless of bone mineral density), and related to age.

 

Eventual bone fracture toughness needs to be determined in mice (half the population at 90% and the remaining at 100% of average life-expectancy) to assess the lifetime effects of a very high (3%), high (1.5%), moderate (0.5%), low (0.2%) and very low calcium diet (0.1%) respectively (Ca/P=1.5, Ca/Mg=10, Mg>0.02%).

 

 

(2) Maybe you can even create 5 groups;

All mice are fed dehydrated beef and fruits (no olives, figs, berries, oranges, kiwi) only, and drinking water. (no nuts because of their high calcium contents)

Calcium will be added to the drinking water of all mice except for the 'very low-calcium' group.

 

The calcium contents of the diet of the 'very low-calcium' group [1], needs to be about 0.01 to 0.015%.

Group [2] (low calcium), needs a little calcium supplementation in the drinking water to create a 0.02% calcium diet.

Group [3] (medium), needs calcium supplementation in the drinking water to create a 0.04% calcium diet.

Group [4] (high-calcium), needs calcium supplementation in the drinking water to create a 0.1% calcium diet.

Group [5] (very high calcium), needs calcium supplementation in the drinking water to create a 1% calcium diet.

 

 

 

 

Q and A about 'the mice calcium-test' (2)

 

 

Does your diet not provide enough calcium according to RDAs? 

Do I need to eat nuts to absorb enough calcium?

Can I meet the RDA for calcium with your diet?

What if I don't buy your theory about calcium and osteoporosis? 

If I'm overweight, a smoker, and don't do sports, do I need more calcium?

What do you think of the calcium RDA?




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Does your diet not provide enough calcium according to RDAs?

With my diet, you CAN consume as much calcium as recommended (according to the calcium RDA). If you want to, you just need to make sure to consume enough nuts. (submit adapted versions of your version of the diet, after adding more nuts into it, to the nutrient calculator , until you have met all RDAs)

 

 

 

 

 

 


"Do I need to eat nuts to absorb enough calcium?"

 

That depends on whether you believe in the calcium-RDA, or my theory about calcium & osteoporosis.

If you believe in the calcium RDA, yes, you need to eat nuts.

But, you need to know that the calcium RDA is based on a hypothesis that has been proven to be wrong; check this out.

It has been shown that the body very effectively adapts the calcium absorption rate accordingly to the availability of calcium in the food that you eat. If consume very little calcium, the absorption rate is much higher, to such an extend that it does not matter whether you consume only 300 or 1,000 mg calcium daily; in both cases about 200 mg is absorbed into the blood. (for proof, check out this site 

This is not a 'mistake' that needs to be corrected by consuming so much calcium that the body can no longer effectively decrease the absorption rate. No, this means that the body does not want to absorb more than 200 mg daily, simply because it only needs to absorb 200 mg daily. And any variety of diet consisting of natural foods only, will already supply you with sufficient calcium.

So, regarding my theory, you don't need to consume nuts to absorb enough calcium, but you do need to consume a little nuts every once in a while to perfectly balance your diet regarding multiple nutrients.

 

 

 

 

 

 

 
"Can I meet the RDA for calcium with your diet?"

 

Yes, by including enough nuts in your diet. 

Submit your version of the diet to the nutrient calculator and compare its total calcium contents with the calcium RDA. If the calcium RDA is not met, simply add more nuts into your diet, and re-submit your (adapted) version of the diet to the nutrient calculator. Repeat this until your diet meets the calcium RDA.

 

 

 

 

 

 

 


"What if I don't buy your theory about calcium and osteoporosis?"

 

If you don't buy my theory but you do believe in the calcium RDA, you better make sure that your version of the diet does meet the calcium RDA. Here's how you can make sure. 

 

 

 

 

 

 


"If I'm overweight, a smoker, and don't do sports, do I need more calcium?"

 

Your need for certain nutrients is elevated if you smoke, or if you live in a big city, but this is not true for the need for calcium.

It is true for nutrients like vitamin C and e because you need more of these antioxidants if you ingest more pro-oxidative chemicals, but calcium has not such an anti-oxidant function.

Actually, calcium's properties are pro-oxidative. 

And...

If you sport, more bone-cells get damaged, due to the higher pressure on the bones, just like you need more dietary protein to replace damaged / worn out muscle cells after having been physically active. So, you actually need more calcium when you do sports.

The role of overweight is dual;

At one hand there is more pressure on the bones if you are overweight, and therefore calcium-bone metabolism will be elevated, but, on the other hand, in obesity the leptin level is elevated (1) and leptin (also) inhibits bone-formation by the osteoblasts. (2) And if bone formation is inhibited, less calcium is required.

 

(1) Chu NF, et al, Plasma leptin concentrations and four-year weight gain among US men. Int. J. Obes. Relat. Metab. Disord. 2001 / 25 (3) / 346-353. , Szymczak E, et al, The role of leptin in human obesity. Med. Wieku. Rozwoj. 2001 / 5 (1) / 17-26. , Hu FB, et al, Leptin concentrations in relation to overall adiposity, fat distribution, and blood pressure in a rural Chinese population. Int. J. Obes. Relat. Metab. Disord. 2001 / 25 (1) / 121-125. , Bahceci M, et al, The effect of high-fat diet on the development of obesity and serum leptin level in rats. Eat. Weight. Disord. 1999 / 4 (3) / 128-132. , Milewicz A, et al, Plasma insulin, cholecystokinin, galanin, neuropeptide Y and leptin levels in obese women with and without type 2 diabetes mellitus. Int. J. Obes. Relat. Metab. Disord. 2000 / 24 / Suppl 2 / S152-3. , Nakamura M, et al, Association between basal serum and leptin levels and changes in abdominal fat distribution during weight loss. J. Atheroscler. Thromb. 2000 / 6 (1) / 28-32. , Bunger L, et al, Leptin levels in lines of mice developed by long-term divergent selection on fat content. Genet`. Res. 1999 / 73 (1) / 37-44.
(2) Burguera B. et al, Leptin reduces ovariectomy-induced bone loss in rats. Endocrinology 2001 / 142 (8) / 3546-3553. , Takeda S, et al, Central control of bone formation. J. Bone Miner. Metab. 2001 / 19 (3) / 195-198. , Anselme K, et al, Comparative study of the in vitro characteristics of osteoblasts from paralytic and non-paralytic children. Spinal Cord 2000 / 38 (10) / 622-629. , Ducy P, et al, Leptin inhibits bone formation through a hypothalamic relay: a central control of bone mass. Cell 2000 / 100 (2) / 197-207.



 

 


 

 


"What do you think of the calcium RDA?" 

 

The calcium RDA is based on the hypothesis that one may prevent osteoporosis by consuming much calcium all your life.

However, statistical data show that hip fracture incidence is the highest in all countries where much calcium is consumed. (check this site for evidence)

To cover up this evidence, they simply changed the definition of osteoporosis, from "porous bones, non-reversible", to "a bone mass lower than recommended". 

But, one can have perfectly healthy bones with a very low bone mass, which is proven by the simple fact that in countries where the average bone bass is lowest, the hip fracture incidence is the lowest too. So, no, a low bone mass is not at all equal to 'sick' / porous bones.

 

The RDA is based on the assumption that a high bone mass may be preventive regarding osteoporosis, which is like reasoning that bodybuilding may prevent muscular diseases.

They solely base their theory on the fact that consuming more calcium adds mass to your bones, and that more bone mass means stronger bones. But, this is only just as true as that working out increases muscle strength, not that this may be preventive regarding muscular diseases.

 

Osteoporosis is a disease, and certainly not comparable (or even equal) to bone mass. 

Actually, consuming more calcium accelerates the aging of bone cells, eventually increasing hip fracture risk, just like sun exposure accelerates aging of skin cells, eventually making you look older sooner. You woun't notice this as long as you are young, because a tan makes you look healthy and vibrant, but as you grow older, you will eventually notice the harmful effects (early dehydration/wrinkling) of excessive sunlight exposure.

The same goes for consuming too much calcium; initially only increasing your bone mass and strength, but also accelerating the aging of bone cells, increasing the risk of exhausted bone cells too early in life; 'real osteoporosis'.

 

So, the calcium RDA is based on an assumption that has been proven wrong. The problem is that they woun't recognize this easily, because it goes against everything they have believed in and claimed for so long, and because than they would have to admit that they have been giving the wrong advices for so many years.

the good news is that your body perfectly knows how much calcium it needs, and that it will always absorb the right amount of calcium, no matter how little / much calcium is consumed, unless FAR too much (over 1,200 mg) or too little (less than 200 mg) dietary calcium is consumed.

Consuming anything between 300 and 800 mg calcium is safe; your body will adapt the absorption rate adequately, according to your calcium intake.

 

 



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